Age-Related Macular Degeneration - download pdf or read online
By D. Virgil Alfaro III et al.
- Learn from a popular workforce of retina surgeons who tackle all points of administration, together with anatomy, body structure, and pathophysiology; imaging of the macula in ARMD, prevention measures and therapy; and destiny instructions within the field.
- Gain a fantastic beginning in intravenous fluorescein angiography, indocyanine eco-friendly angiography, and optical coherence tomography, in addition to antiVEGE treatment and comparable new medicines, optimizing your analysis and therapy options.
- Understand the numerous switch within the category method of choroidal neovascular membranes in ARMD.
- Get up to date info on treating ARMD secondary to geographic atrophy via cellphone transplantation and pharmacologic therapies.
- Easily visualize key features of ARMD because of high quality illustrations and photographs.
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Extra resources for Age-Related Macular Degeneration
Eyes of aged mice expressing human ApoE2, ApoE3, or ApoE4 and maintained on a high fat and cholesterol (HF-C) diet showed ApoE isoform–dependent pathologies of diverse severity. They develop a constellation of changes that mimic the pathology associated with human AMD such as ■■ Diffuse subretinal pigment epithelial deposits ■■ Drusenoid deposits ■■ Thickened BM ■■ Atrophy, hypopigmentation, and hyperpigmentation of the RPE Interestingly, ApoE4 mice were the most severely affected, to the point of having sometimes developed CNV.
Glycation-altered proteolysis is accounted for linking dietary GI, aging, and agerelated diseases like AMD. Not surprisingly, consuming higher GI diets was associated with over threefold higher accumulation of advanced glycation end products (AGEs) in the retina in the age-matched mice. Both incidence and severity of retinal changes aggravated with age, as older mice exhibited more thickening of the BM, more basal laminar deposits, disorganization of RPE basal infolding, and increased photoreceptor atrophy compared to 17-monthold counterparts (32).
235) used rabbits in an attempt to find an intermediate laser-induced CNV model between rodents and primates, thereby avoiding the expense and ethical issues concerning the latter. Nonetheless, rabbits exhibit significant anatomical discrepancies, as they do not have a macula and their retina vascular supply is different. The model utilized subretinal endophotocoagulation to create histologically identified CNV. Pig Another intermediate laser-induced CNV model is the pig. Saishin et al. (219) used a laser to create defects in the BM in the pig eye and establish histologic evidence of CNV in 100% of lesions.
Age-Related Macular Degeneration by D. Virgil Alfaro III et al.